This triad of symptoms results from a deficiency in vitamin B 1 which is an essential coenzyme. WE is characterized by the presence of a triad of symptoms: It involves an acute Wernicke-encephalopathy phase, followed by the development of a Korsakoff syndrome phase. The syndrome is a combined manifestation of two namesake disorders, Wernicke encephalopathy and alcoholic Korsakoff syndrome. Of those affected, 25% require long-term institutionalization in order to receive effective care. Failure in diagnosis of WE and thus treatment of the disease leads to death in approximately 20% of cases, while 75% are left with permanent brain damage associated with WKS. Wernicke encephalopathy and WKS are most commonly seen in people with an alcohol use disorder. WKS is usually secondary to prolonged alcohol abuse. These disorders may manifest together or separately. This can occur due to Wernicke encephalopathy, eating disorders, malnutrition, and alcohol abuse. The cause of the disorder is thiamine (vitamin B 1) deficiency. It mainly causes vision changes, ataxia and impaired memory. Due to the close relationship between these two disorders, people with either are usually diagnosed with WKS as a single syndrome. Wernicke–Korsakoff syndrome ( WKS) is the combined presence of Wernicke encephalopathy (WE) and Korsakoff syndrome. Korsakoff's psychosis, alcoholic encephalopathy, "wet brain" Medical condition Wernicke–Korsakoff syndrome
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